The analysis of wild-type C. elegans and 9 mutant strains revealed that sulforaphane inhibited DAF-2 insulin/insulin receptor signaling and thereby increased DAF-16 nuclear translocation ...

Different wild-type strains of C. elegans vary in their tendency to leave or remain on a standardized small lawn of bacterial food ().For example, adult hermaphrodites from the laboratory strain ...

A wild-type worm moving across the bottom of a petri dish. Eugene Lee/MIT A C. elegans mutant incapable of laying eggs properly as seen through a blue filter. Eugene Lee/MIT.

Scanning electron microscopy images of the cuticle (skin) of C. elegans kept at 25 degrees Celsius show wrinkles on an old (9-day old) adult wild-type worm (left) but smooth skin on an npr-8 ...

In well-fed wild-type animals, DAF-16 was present in the cytoplasm with a C:N ratio of 1.2; however, in daf-2 mutants, DAF-16 was translocated to the nucleus with a C:N ratio of 0.5.

However, whether and how centrosomal RNAs exist in C. elegans and, if yes, their functions and mechanisms remain unclear. ... Left: NRDE-3 localizes in nucleus in wild-type background.

They then tested the drug on C. elegans and found that it increased wild-type worm lifespan. Additionally, they found that metolazone did not extend lifespans in worms in whom the genes atfs-1, ubl-5, ...

Pattern recognition system that monitors disease-causing bacteria in C. elegans A nuclear hormone receptor intercepts pathogen-derived signals of growth and virulence, revealing an evolutionarily ...

In this behavior, C. elegans kept in an environment with food at 23 degrees Celsius will move toward 23 degrees when placed in an environment with a temperature gradient from 17 to 23 degrees.

Oregon Health & Science University, in collaboration with Oregon State University, has discovered the structural organization and protein components of a lipid-transfer complex known as LPD-3.

Researchers demonstrated that the microscopic worm C. elegans uses a retrotransposon called Cer1 to transfer a learned behavior (avoidance of a pathogenic bacterium) between worms.